Author(s): Kênia M. da Paula | Paula B. Viegas | Paula G. Silva
Journal: Revista de Biologia e Ciências da Terra
ISSN 1519-5228
Volume: 2;
Issue: 2;
Date: 2002;
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Keywords: p53 | macrophages | necrosis | ICE-proteases | lymphocytes T | timocits | Bcl-2 | apoptosis
ABSTRACT
The apoptosis, where occur the death cellular programmer, many times is one benefice for the organism, serving as a mechanism of defense. The cell accept a signal for herself shatter, shrink of size, rupture at chromatin in pieces and herself easily phagocytes. The death by apoptosis no provoke at inflame. On the contrary, at necrosis, death cellular usually by absence of oxygen, by microorganism pathogenic or by substance toxic get what them cells herself break and cast your contents to out of cell promoting response inflammatory us goods neighbors. Them cells and the organelles increase of bulk occurring to break your cell.
Journal: Revista de Biologia e Ciências da Terra
ISSN 1519-5228
Volume: 2;
Issue: 2;
Date: 2002;
VIEW PDF
DOWNLOAD PDF Original pageKeywords: p53 | macrophages | necrosis | ICE-proteases | lymphocytes T | timocits | Bcl-2 | apoptosis
ABSTRACT
The apoptosis, where occur the death cellular programmer, many times is one benefice for the organism, serving as a mechanism of defense. The cell accept a signal for herself shatter, shrink of size, rupture at chromatin in pieces and herself easily phagocytes. The death by apoptosis no provoke at inflame. On the contrary, at necrosis, death cellular usually by absence of oxygen, by microorganism pathogenic or by substance toxic get what them cells herself break and cast your contents to out of cell promoting response inflammatory us goods neighbors. Them cells and the organelles increase of bulk occurring to break your cell.
